DR S WK 8
The patient is a 54 years old man with a complete loss of legmovements, which occurred abruptly. No trauma is directly linked tothe symptoms. Additionally, the patient has lost sensations of painand temperature, bilaterally below T9. There is no numbness observedor reported above T9. Other senses such as localized touch,vibrations and position are normal. Motor system analysis indicatesthat the patient has a bilateral paralysis below T9, but other partsare healthy (Lundy-Ekman, 2007).
Based on the signs and symptoms observed, the frontal and superiormedial parietal lobes of the brain are affected. Therefore, lesionsare likely to be found in the anterior cerebral artery. Occlusion ofthe blood vessels could have resulted in hemiplegia and hemisensoryloss, which is severe in the lower limbs. This is due to its effectson the white matter and sensorimotor cortex in the frontal lobe. Thelack of blood supply to the tissues harms the anterior putamen andother structures, resulting in motor dysfunction. This is unlikely toaffect autonomic functions or special senses, but may have impacts onemotions and behavior, urinary incontinence, and cognitive abilities(Lundy-Ekman, 2007).
The condition is caused by the occlusion of the anterior cerebralartery, which can be as a result of a wide range of factors. Forexample, it may occur due to emboli, associated with narrowing of themajor blood vessels or a combination of thrombosis andatherosclerotic stenosis. In some cases, it can also be induced bylipohyalinotic conditions. However, the symptoms of the disease areonly observed when the blockage occurs before the anteriorcommunicating artery. This is due to collateral circulation processesin the brain. Otherwise, the symptoms will be very mild (O`Sullivan,2014).
Lundy-Ekman, L. (2007). Neuroscience: Fundamentals forrehabilitation. St. Louis, MO: Saunders/Elsevier.
O`Sullivan, S. (2014). Physical rehabilitation. Philadelphia:F.A. Davis Co.